Learning disabilities in children and young people 

An outline of learning disabilities

Learning disabilities (LD) are estimated to occur in between 5 - 8% of schoolage children1,3,5,6 and are defined as the presence of a significant learning problem in one or more of the three learning areas of reading, writing and mathematics, with academic achievement in these areas falling below that of the average expected of the child’s particular age and intellectual capacity.1,3 Beyond the general diagnosis of LD are the specific disorders of learning in the areas of reading (dyslexia), maths (dyscalculia), writing (dysgraphia) and motor problems (dyspraxia).7 Future outcomes for children diagnosed with LD largely concern academic and employment underachievement but can also include an increased likelihood of developing emotional and behavioural problems, brushes with the law, and exhibiting anti-social behaviour, although these are largely thought to be more a result of potential co-occurring disorders such as ADHD.4,8,9,10,11,12,13,14 However, the definiton of learning disabiities is still one of the most hotly debated issues surrounding the topic itself, stemming from the way in which diagnosis of LD is made.1,2,3,5,15

Traditionally, and still officially, the main method of diagnosis is the IQdiscrepancy model, where a diagnosis of LD is made when the academic achievement of the child is below the average expected for a child with the same intelligence quotient score (IQ).3,6,7 However, this method alone involves a ‘wait-to-fail’ approach, in that the problem is often identified late on, when difficulties have started to accumulate.1,5 Given these problems, the current trend of assessment, diagnosis and treatment of LD is moving toward a Response-to-Intervention model (RTI).1,5,6,16,17,18,19 Through this model, children with difficulties are identified and treated early, with only the most persistent of problems, leading to a diagnosis of LD.1,5,16,17,18,19,20,21

Assessment and diagnosis of LD can, therefore, involve a number of professionals, depending on the suspected problem of the child. For example, it could be that poor eye movement is the cause of a deficit in reading skills and thus, would require the necessary consultation with an eye specialist to assess the child’s occulomotor functioning.1 However, such assessments are carried out to aid the overall diagnosis, rather than diagnose per se, with diagnosis typically being performed by an educational psychologist.6

 

Theories and causes

Theories as to why LD occur can be divided into two main areas: neurological theories and psychological theories.

Neurological theories of LD have their origin in the observation that those disorders with a definite genetic or neurological causal factor also present LD, such as the genetic-based Down’s syndrome and Fragile X syndrome, or Fetal Alcohol Syndrome, where the mother’s consumption of alcohol during pregnancy causes neurological damage to the child.22,23,24,25,26 However, although the search for a genetic cause of LD in milder cases outside of these specific disorders has implicated some genetic and neurological mechanisms, researchers have yet to find a definite neurological cause. Much of the reason for this is attributed to the heterogeneity of the disorder, taken form the seven more specific symptom areas where reading, writing and maths deficits can occur (see box 1).

Box 1: Symptoms 3,22

Symptoms of LD include difficulties with:

  1. speaking, such as difficulty in pronouncing words and forming sentences
  2. listening, such as being unable to follow and re-tell stories
  3. basic reading skills, such as word recognition or pronouncing simple pseudo-words
  4. reading comprehension, such as understanding a written story
  5. simple arithmetic calculation, such as 2 + 2 = 4
  6. mathematical reasoning, such as following counting principles (e.g. counting in twos starting from 0 = 0, 2, 4, 6 etc)
  7. written expression, such as writing simple stories.

Psychological theories, on other hand, take a more direct approach toward the causes of LD by focusing on the underlying cognitive processes that are taken to give rise to the problems faced by children with LD.28,29,30 Some of the cognitive processes involved here include phonological awareness and working memory (see box 2).31,32,33 However, although some interesting work has broadened the understanding of the psychological causes of LD, again, the heterogeneity of LD means that the relationship between specific underlying cognitive deficits and the variety of problems experienced is not yet fully understood.1,3

Although the search for underlying and neurological causes has not et provided a full understanding as to how and why LD develop, an encouraging recent trend has developed toward a RTI approach, not only for diagnosis, but also for treatment.5,6,16,17,18,19,20,21

Box 2: Cognitive Processes

Examples of some of the cognitive processes that may be involved in LD include:

Working memory – works by temporally storing information to be used on the task at hand, such as the numbers at each stage of a mathematical calculation.

Phonological awareness – essential in the understanding of the relationship between written letters/words and sounds. Problems in mapping words to sounds can be responsible for poor reading skills.

Semantic memory – concerns the memory for meanings, definitions, understandings and other concept-based knowledge. A deficit in semantic memory for a child with LD may mean that they cannot remember the meanings of grammatical rules or mathematical concepts.

 

Treatment, support and management, and the Response-to Intervention (RTI) framework

Treatment, support and management for LD typically centre on a RTI approach. Developed under a framework aimed at ensuring the effectiveness of both the general and special educational settings in providing children with the necessary opportunities to learn, the RTI model focuses on early identification and treatment for children experiencing learning problems.5,16,17,19

The first stage of the RTI model involves identification. This may take the form of the more traditional approach to the field, namely where academic achievement falls significantly below the average for a child’s age and IQ. However, the emphasis here is on a much earlier assessment and identification of difficulties across the whole classroom.5,6,19

The second stage of the RTI model then makes provisions for those children experiencing some learning problems. These provisions commonly take the form of small tutorial groups that use specific techniques for further assessment.5,6,19

The third stage of the RTI model can then operate in one of two ways; children who respond to the provisions made and whose difficulties improveare then deemed “disability free” and returned to the general educational setting, whilst those with more persistent problems are referred for further assessment and considered for special education.5,6,19

Support for LD may come in a variety of formats, given the multiple ways in which a LD can manifest.6,34 For example, some children may benefit from small teaching groups that utilise rigid and highly structured lesson plans with a focus on their specific needs. In addition, the provision of classroom aids may be required, such as computer software designed to provide reading, writing and maths learning assistance. Beyond the education setting however, there are also a number of support groups and resources available to children with LD and their families.35,36,37,38,39,40

Advantages of the RTI model are the avoidance of a ‘wait-to-fail’ situation, where diagnosis and treatment only occur when a problem becomes explicit,usually around 8 or 9 years old, and treatment at this stage and beyond is less effective.1,6 The pre-school intervention however, indicates the effectiveness and dedication of the RTI model toward early identification and treatment.

Another encouraging approach toward the topic of LD is that of risk and resiliency (RR),42,43,44,45,46,47,48 whereby research attempts to identify those risk factors that may lead to a child with LD developing more significant problems as they get older, such as substance abuse, and the resiliency factors that may steer children away from such outcomes.42,43,44,45,46,47,48

Conclusions

Although the very definition, diagnosis and existence of LD are still widely ebated topic areas1,2,3, there is no doubt that there are a significant proportion of children who experience a very real problem in one of more of the areas of reading, writing and maths. Moreover, in the absence of early identification and treatment of LD, the outcome for children with a LD diagnosis can be disappointing, with an increased likelihood toward poor academic achievement, employment status and emotional or behavioural problems.4 Causal theories of LD have also failed to provide a clear and definite underlying cause however,1 although some neurological and psychological theories have been put forward.

However, the current stance in this field is far from a negative one, with a focus on the early identification and intervention, a thriving approach.5,6 As a result, there is a wide variety of support available for children at risk, or diagnosed with, a LD and their families. Similarly, research, governmental policies, professional protocols, and RTI treatment approaches, all converge to provide a focus on overcoming the difficulties that a LD diagnosis may bring, and seek to improve the services and long-term outcomes for children that develop LD

Although there are still some issues that require some attention, such as the nature of diagnosis, the current status of the LD field is an encouraging one, with a continued positive approach to early identification, treatment, and further understanding of these areas, and LD in general, forthcoming.

References

  1. Lyon, G. R. 1996. Learning disabilities. The Future of Children, 6, 54–76
  2. Fletcher, J. M., Morris, R. D. & Lyon, G. R. 2005. Classification and defintion of Learning disabilities: An integratvie perspective. In Swanson, H. L., Harris, K. R. & Graham, S. (eds) Handbook of learning disabilities. New York, The Guldford Press. P. 30-56
  3. Lyon, G.R., Fletcher, J.M., Shaywitz, S.E., Shaywitz, B.A., Torgesen, J.K., Wood, F.B., Schulte, A. & Olson, R. 2001. Rethinking learning disabilities. In rethinking special education for a new century. C.E. Finn, A.J. Rotherham & C.R. Hokanson (Eds.) Washington, D.C.: Thomas B. Fordham Foundation and the Progressive Policy Institute
  4. Johnson, B. 2005. Psychological co-morbidity in children and adolescents with learning disorders. J Indian assoc child adol mental health, 1, 1-7
  5. Vaughn, S. & Fuchs, L. S. 2003. Redefining learning disabilities as inadequate response to instruction: The promise and potential problems. Learn Disabil Research and Prac, 18, 137-146
  6. Department for Education and Employment. 1997. Excellence for all children: meeting special education need. London, DfEE
  7. American Psychiatric Association. 2000. Diagnostic and statistical manual of mental disorders. Fourth edition, revised 2000. Washington DC: Author
  8. Willcutt, E. G. & Pennington, B. F. 2000a. Comorbidity of reading disability and attention-deficit/hyperactivity disorder: differences by gender and subtype. J Learn Disabil, 33, 179-91
  9. Willcutt, E. G. & Pennington, B. F. 2000b. Psychiatric comorbidity in children and adolescents with reading disability. J Child Psychol Psychiatry, 41, 1039-48
  10. Seager, M.C. O’Brien, G. 2003. Attention deficit hyperactivity disorder: Review of ADHD in learning disability: The diagnostic criteria for psychiatric disorders for use with learningdisabilities/mental retardation(DC-LD) criteria for diagnosis, J Intellectual Dis Res, 47, 26-31
  11. McGillIvray, J. A. & Baker, K. L. 2009. Effects of comorbid ADHD with learning disabilities on anxiety, depression, and aggression in adults. J Atten Disord, 12, 525-31
  12. Karakas, S. Turgut, S. & Bakar, E. E. 2008. Neuropsychometric comparison of children with “pure” learning disabilities, “pure” ADHD, co-morbid ADHD with learning disabilities and normal controls using the Mangina-Test (analytical-specific visual perception). Symposium abstracts/Int J psychophysiology, 69, 139-205
  13. Kshani, J.H., Cantwell, D.P., Shekim, W.D. & Reid, J.C. 1982. Major depressive disorder in children admitted to inpatient community mental health centre. American Journal of Psychiatry, 139, 671–672
  14. Prior, M., Sanson, A., Smart, D. & Oberklaid, F. 1999. Relationships between learning difficulties and psychological problems in preadolescent children from a longitudinal sample. Journal of American Academy of Child Adolescent Psychiatry, 36, 1020-1032
  15. Lyon, G.R. 1995. Toward a definition of dyslexia. Annals of Dyslexia, 45, 3–27
  16. Fuchs, L. S. 1995. Curriculum-based measurement and eligibility decision making: An emphasis on treatment validity and growth. Paper presented at the Workshop on Alternatives to IQ Testing. Washington, DC: National Academy of Sciences
  17. Fuchs, L. S., & Fuchs, D. 1998. Treatment validity: A unifying concept for reconceptualising the identification of learning disabilities. Learning Disabilities Research & Practice, 13, 204–219
  18. Gresham, F. M. 2002. Responsiveness to intervention: An alternative approach to the identification of learning disabilities. In R. Bradley, L. Danielson, & D. P. Hallahan (Eds.), Identification of learning disabilities: Response to treatment (pp. 467-519). Mahwah, NJ: Erlbaum
  19. Heller, K. A., Holtzman, W. H. & Messick, S. 1982. Placing children in special education : a strategy for equity, Washington, D.C, National Academy Press
  20. Chard, D. J., Vaughn, S. & Tyler, B. J. 2002. A synthesis of research on effective interventions for building reading fluency with elementary students with learning disabilities. J Learn Disabil, 35, 386-406
  21. Donovan, M. & Cross, C. 2002. (eds). Minority students in special and gifted education, committee on minority representation in special education, Washingto D. C.
  22. Alberman, E. 1978. Main causes of major mental handicap: prevalence and epidemiology. Ciba Found Symp, 3-16
  23. Dykens, E., Leckman, J., Paul, R. & Watson, M. 1988. Cognitive, behavioral, and adaptive functioning in fragile X and non-fragile X retarded men. J Autism Dev Disord, 18, 41-52
  24. Jones, K. L., Smith, D. W., Ulleland, C. N. & Streissguth, P. 1973. Pattern of malformation in offspring of chronic alcoholic mothers. Lancet, 1, 1267-71
  25. Raymond, F. L. & Tarpey, P. 2006. The genetics of mental retardation. Hum Mol Genet, 15 Spec No 2, R110-6
  26. Sue, D., Sue, D. & Sue, S. 1994. Understanding Abnormal behaviour (4th edition). Boston:Houghton Mifflin
  27. Carpenter, P. 1997. Learning Disability. In Rees, L., Lipsedge, M. & Ball, C. (eds) Textbook of psychiatry. London: Arnold
  28. Sanovich, K. E. 1986. Matthew effects in reading: Some consequences of individual differences in the acquisition of literacy. Reading Research Quarterly, 21, 360-407
  29. Stanovich, K. E., & Siegal, L. S. 1994. Phenotypic performance profiles of children with reading disabilities: A regression based test of the phonological-core variable difference model. Journal of Educational Psychology, 86, 24–53
  30. Egan, J. 1998. Why can’t dyslexics read? Psychol review, 5, 12-15
  31. Ramus, F., Rosen, S., Dakin, S. C., Day, B. L., Castellote, J. M., White, S. & Frith, U. 2003. Theories of developmental dyslexia: insights from a multiple case study of dyslexic adults. Brain, 126, 841-65
  32. Fletcher J.M., Shaywitz, S.E., Shankweiler, D.P., Katz, L., Liberman, I.Y., STUEBING, K.K., FLETCHER, J.M., SHAYWITZ, S.E., & SHAYWITZ, B.A. 1994. Co-morbidity of learning and attention disorders: Separate but equal. Pediatric Clinics of North America, 46, 885–897
  33. Adams, M.J. 1990. Beginning to read: Thinking and learning about print. Cambridge, MA: MIT Press
  34. Maretsen, D., Muyskens, P., Lau, M. & Canter, A. 2003. Problem solving model for decision making for high incidence disabilities: The Minneapolis experience, Learning disabilities research and practice, 18, 187-200
  35. Contact a Family [online]. Available from http://www.cafamily.org.uk/ [Accessed 15 December 2009]
  36. Enable [online]. Available from http://www.enable.org.uk/index.php [Accessed 15 December2009
  37. Department of Health [online]. Available from http://www.dh.gov.uk/en/SocialCare/Deliveringadultsocialcare/Learningdisabilities/index.htm [Accessed 15 December 2009]
  38. Learning Disabilities Wales [online]. Available from http://www.learningdisabilitywales.org.uk/about.php [Accessed 15 December 2009]
  39. NHS Evidence [online]. Available from http://www.library.nhs.uk/learningdisabilities/2 [Accessed 15 December 2009]
  40. Joseph Rowntree Foundation [online]. Available from http://www.jrf.org.uk/about-us/contact-details [Accessed 15 December 2009]
  41. Star [online]. Available from http://www.sthelens.gov.uk/lgnl.htm?id=9 [Accessed 15 December 2009]
  42. Morrison, G. M. & Cosden, M.A. 1997. Risk, resilience, and adjustment of individuals with learning disabilities. Learn dis quart, 20, 43-60
  43. Werner, E. E. (Ed.). 1986. A longitudinal study of perinatal risk. Orlando, FL: Academic Press
  44. Werner, E. E. 1993. Risk and resilience in individuals with learning disabilities: Lessons learned from Kauai longitudinal study. Learning disabilities research and practice, 8, 28-34
  45. Werner, E. E. & Smith, R. S. 1982. Vulnerable but invincible: A longitudinal study of resilient children and youth. New York: McGrath-Hill
  46. Werner, E. E. & Smith, R. S. 1992. Overcoming the odds: High risk children from birth to adulthood. Ithaca, NY: Cornell University Press
  47. Werner, E. E. & Smith, R. S. 2001. Journeys from childhood to midlife: Risk, resilience and recovery. Ithaca, NY: Cornell University Press
  48. Wong, B. Y. L. 2003. General and specific issues for researchers consideration in applying the risk and resilience framework to the social domain of leanring disabilities. Learning disabilities research and practice, 18, 68-72

This information is not meant to replace the advice of any physician or qualified health professional. The information provided by Cerebra is for information purposes only and is not a substitute for medical advice or treatment for any medical condition. You should promptly seek professional medical assistance if you have concerns regarding any health issue.

 

Page last updated: 21/12/2011 15:40 
 
© 2012 Cerebra | Registered charity No: 1089812 | Company Limited by Guarantee, registered in England and Wales 4336208